| Genetic tests exist for Celiac disease and are highly | | | | The most commonly used laboratories for celiac |
| accurate for determining the risk of the disease. | | | | disease genetic testing in the U.S. are Kimball |
| When a complete genetic panel is performed the | | | | Genetics, LabCorp, Quest, Prometheus, and Enterolab. |
| possibility that someone having or ever getting this | | | | The Laboratory at Bonfils in Denver not only |
| autoimmune disease can be determined to an | | | | provides testing directly but also does the testing for |
| extremely high degree of certainty. Unfortunately, | | | | several hospitals, Quest and Enterolab. Bonfils only |
| some tests are misleading because they do not | | | | does beta subunit testing. They report results of |
| include a portion of the genetic pattern that may be | | | | DQ2 and DQ8 negative based on the absence of the |
| present that can predispose to this gluten sensitivity | | | | beta subunits associated with DQ2 and DQ8. |
| disease though the report may imply absence of | | | | However this is somewhat misleading since someone |
| increased risk. | | | | could have only the alpha subunit and be "partially" |
| Some genetic tests can be done without a doctor's | | | | DQ2. |
| order. Insurance coverage for the Celiac genetics is | | | | Though the risk of being "half" DQ2 positive from |
| highly variable. A couple of laboratories can run the | | | | only having the alpha subunit is low overall it is still |
| tests on samples obtained from a mouth swab that | | | | there. Furthermore, there are people who may |
| is painless and well accepted by children. Genetic | | | | believe that they are DQ2 or DQ8 negative based on |
| testing can be done at any age while blood tests for | | | | testing from Bonfils, Quest or Enterolab. These |
| Celiac are not recommended before a year of age. | | | | people and/or their doctor may exclude the possibility |
| Celiac genetic tests are not affected by eating gluten | | | | that they have or are at risk for ever getting Celiac |
| or not. | | | | disease when in fact this may or may not be true. |
| If you do not have the commonly recognized HLA | | | | The existence of DQ2 and DQ8 negative Celiac |
| genetic patterns DQ2 or DQ8 that are associated | | | | disease has been debated. It is probably clouded to |
| with Celiac disease you are believed to not be at risk | | | | some degree by this confusion about the genetics. |
| for the full autoimmune disease. You don't need to | | | | Most experts assert that the presence of DQ2 or |
| be periodically retested. However, you still could be | | | | DQ8 is a requirement to develop the disease and |
| intolerant or sensitive to gluten. Knowing your | | | | their absence excludes the possibility. However, |
| genetics can be very helpful if you have a family | | | | reports of DQ2 and DQ8 negative Celiac disease |
| member with Celiac disease or they or you have | | | | persist. |
| other autoimmune diseases associated with a risk of | | | | I have a couple of patients who have the positive |
| Celiac. | | | | results for the specific blood tests for CD, |
| HLA DQ2 and DQ8 are the simple designations for | | | | endomysial or tissue transglutaminase antibody; and |
| complex white blood cell patterns or types that are | | | | classic biopsy features but were reported DQ2 and |
| known to be associated with an increase risk of | | | | DQ8 negative by laboratories who only test for the |
| Celiac disease. The HLA term stands for human | | | | beta subunit. Ideally, they should be re-testing for |
| leukocyte antigen. Leukocytes are white blood cells. | | | | alpha unit positive "half" DQ2 or DQ8 but this will |
| Antigens are proteins that serve or elicit an immune | | | | depend on their insurance coverage. In the meantime, |
| response by the body. So, the HLA system is a | | | | I am remain concerned that many patients and |
| complex set of proteins on the surface of white | | | | doctors may be lulled into a false sense of security |
| blood cells. Everyone has two copies of a DQ protein | | | | by negative genetic tests incompletely done or that |
| pattern. You get one copy of DQ from your mom | | | | diagnoses of Celiac disease may be or have been |
| and one from your dad. Having at least one copy of | | | | withdrawn on some individuals based on incomplete |
| either is necessary and sufficient to develop the | | | | genetic results. |
| disease. Having two copies of either or one of both | | | | This issue of DQ2 and DQ8 testing is further |
| increases the risk even more. | | | | complicated by reviews on the subject that are |
| These protein patterns are inherited just like the red | | | | incomplete or vague. The best reviews I have found |
| blood cell proteins that constitute what is commonly | | | | are by Ludvig Sollid and Benedicte Lie of Oslo, |
| known your "blood type". I, for example, am A | | | | Norway "Celiac Genetics: Current Concepts and |
| positive blood type. This means I have a pattern of | | | | Practical Applications" Clinical Gastroenterology and |
| proteins designated A and Rh+ on the surface of my | | | | Hepatology 2005 and Bourgey's 2007 review. In a |
| red blood cells. On the other hand I have a white | | | | recent update article by Victorien, there is a general |
| blood cell type pattern DQ2/DQ7 inherited from my | | | | review the genetics of celiac disease including the |
| parents. My Dad gave me a DQ2 and my Mom the | | | | association of myosin IXB gene (MYO9B). However, |
| DQ7. You have two DQ patterns on your white | | | | it doesn't explain the DQ2 or DQ8 typing well. They |
| blood cells that you received from your parents and | | | | conclude that "To date, only HLA-DQ2 or HLA-DQ8 |
| you give one of your DQ types to each of your | | | | typing is clinically relevant..." but fail to point out that |
| children. | | | | HLA DQ2 and DQ8 typing should include both alpha |
| Since only a single copy of either DQ2 or DQ8 can be | | | | and beta subunits. |
| associated with an increase risk of developing Celiac | | | | It is clear that both HLA and non-HLA genetic factors |
| disease, most laboratories test for the presence of | | | | are important in the risk of Celiac disease. However, |
| either and simply report their presence or absence. | | | | the absence of the high-risk genes does not preclude |
| However, knowing if you have one or two copies | | | | adverse reactions to gluten including leaky gut, skin, |
| not only provides additional information about degree | | | | digestive and neurological symptoms. When genetic |
| of your risk. It also may predict the severity. It also | | | | testing is used to try to assess the risk or exclude |
| provides information about your parents and your | | | | CD then I recommend that full testing including both |
| childrens' risk of inheriting an at risk gene. If you have | | | | alpha and beta subunit typing. Hopefully more |
| DQ2 and DQ8 we know your complete DQ pattern. | | | | research will better define the genetics of both Celiac |
| We also known one of your parents had at least | | | | disease as well as non-celiac gluten sensitivity or the |
| DQ2 and the other DQ8. All of your children will either | | | | so called "gluten syndrome". |
| get a DQ2 or a DQ8. So, both your parents and all of | | | | Selected References: |
| your children are at risk for Celiac in that situation. If | | | | Bourgey, M et al. HLA related genetic risk for Coeliac |
| you have only copy of DQ2 or DQ8 then we only | | | | disease. Gut 2007; 56:1054-1059. |
| know that at least one of your parents had one | | | | Johnson, TC et al. Relationship of HLA-DQ8 and |
| copy of the risk gene and each of your children will | | | | severity of Celiac disease: Comparison of New York |
| have a 50-50 chance of inheriting such a risk gene | | | | and Parisian cohorts. Clin Gastroenterol Hep 2004; |
| from you. | | | | 2:888-894. |
| Other non-HLA genetic factors are involved in the | | | | Kaukinen K. et al. HLA-DQ typing in the diagnosis of |
| risk of celiac disease. These are still being worked | | | | Celiac disease. Am J Gastroenterol 2002; 97(3): |
| out. However, one poorly understood and little | | | | 695-699. |
| known fact to most doctors and almost all patients is | | | | Lundin, KE. HLA-DQ8 as an Ir gene in Coeliac disease. |
| that HLA DQ2 and DQ8 testing done by some | | | | Gut 2003; 52:7-8 |
| laboratories does not include the full spectrum of at | | | | Mazzarella G. et al. An immunodominant DQ8 |
| risk components of these patterns. DQ2 and DQ8 | | | | restricted gliadin peptide activates small intestine |
| are a summary blood type designations or serotypes | | | | immune response in in vitro cultured mucosa from |
| for the presence of several protein subunits. There | | | | HLA-DQ8 positive but not HLA-DQ8 negative Coeliac |
| are alpha and beta subunits to these protein patterns. | | | | patients. Gut 2003; 52:57-62. |
| The beta subunit is the most influential and important | | | | Sollid, LM and Lie, BA. Celiac disease genetics: Current |
| component. Most laboratories only test for and | | | | concepts and practical applications. Clin Gastro Hep |
| report the beta subunit. However, the alpha subunit | | | | 2005; 3:843-851. |
| does carry risk on its own, albeit much less than the | | | | Wolters,VM and Wijenga C. Genetic background of |
| presence of the beta subunit or the presence of | | | | celiac disease and its clinical applications. Am J |
| both alpha and beta subunit. | | | | Gastroenterol 2008; 103:190-195. |