The Cholesterol Conspiracy - The Truth About Statins And Nutritional Supplementation

"All truth passes through three stages.considered to be normal, the more people
First, it is ridiculed.automatically become victims of the dreaded disease
Second, it is violently opposed.of "high cholesterol." Therefore, more people will be
Third, it is accepted as being self-evident."persuaded that they need to be taking a statin drug,
Arthur Schopenhauerand voilà, more profit for the manufacturers.
(1788 - 1860)When you consider the size of the profits already
What is the true cause of heart disease, and howreceived, let alone the potential profit from statin
can we truly reduce the risk of death?drugs over the next several years, the cholesterol
Atherosclerosis, or Coronary Artery Disease (CAD), isconspiracy is one of the largest money making
the leading cause of death in both men and women.schemes ever perpetrated on the world.
In the U.S. alone, there are more than one millionIn July 2004, the level of LDL cholesterol considered
heart attacks every year, one third of them resultingnormal underwent another change. The new norm
in death. The majority of men and women currentlyplunged from 100 to 70, virtually doubling again the
have, or are actively developing, atherosclerosis. Bynumber of people who are "infected" with the plague
age 20, most people already have a 15-25%of high cholesterol. Why, it's the epidemic of our time!
narrowing of their arteries due to plaque formation.Many enlightened people howled at this news,
By age 40, there is a 30-50% clogging of theirwondering if the masses would ever wake up and
arteries.see who is behind this, and why. Why is the medical
In the beginning of the Twentieth Century,establishment ignoring the thousands of published
congestive heart disease (CHD) was mostly a resultmedical studies that show the beneficial effects of
of rheumatic fever, which was a childhood disease.nutritional supplements against heart disease? Why is
However by the year 1936 there was a dramaticthe medical establishment down-playing the
change in the main cause of heart disease.dangerous and deadly side effects of statin drugs?
Cardiovascular disease caused by atherosclerosis, orThe "updated" LDL cholesterol recommendations
plaque buildup, took first place as the primary causewere published in the July 2004 issue of the
of heart disease, making congestive heart failure aAmerican Heart Association's publication, Circulation. A
distant second.panel from the National Heart, Lung and Blood
During the 1950's, the autopsies conducted on menInstitute, a division of the National Institutes of
who died of heart disease that revealedHealth, which is endorsed by the American College of
plaque-clogged arteries concluded that cholesterolCardiology, and the American Heart Association, were
was the cause of hardening of the arteriesthe ones who actually pronounced the new
(atherosclerosis) and coronary artery disease.cholesterol level at which drugs should be prescribed.
Cholesterol, not calcium, was considered the "cause"Sounds pretty official and reliable if these powerful
of heart disease, despite plaque consisting of 95%medical institutions are backing up these
calcium and a relatively small percentage ofrecommendations, right?
cholesterol. By 1956 there were 600,000 deathsThe fact is eight of the nine panel members making
annually from heart disease in the U.S. Of thosethe new LDL cholesterol recommendations were
600,000, 90% were caused by atherosclerosis, orbeing paid by the statin-producing pharmaceutical
clogged arteries. In fewer than 25 years, the numbercompanies. The panelists did not disclose their financial
one cause of death in the U.S. had changedconflict of interest. This information was uncovered
dramatically ...from congestive heart disease toby Newsday, a Long Island, New Yorknewspaper (D.
coronary artery disease.Ricks and R. Robins, Newsday, July 15, 2004). Seven
Because cholesterol was dubbed the "cause" ofof the nine panelists have financial connections to
atherosclerosis, the effort to lower cholesterol byPfizer, the makers of Lipitor®. Five of the nine
any means began in earnest. Both the food industryserved as "consultants" to Pfizer. So, what did the
and the pharmaceutical industry seized upon thisother two panelists do to deserve their money?
opportunity to cash in on a cholesterol-loweringSeven of the nine panelists also received money
campaign by creating foods and drugs that wouldfrom Merck, the producers of Zocor®, with four
supposedly save lives. Diets, such as the Prudentof them serving as "consultants" to the company.
Diet, were established to lower the amount ofEight of the panelists who made the
cholesterol intake from food. There was no doubtrecommendations that would increase the prescribing
that both polyunsaturated oils and drugs reducedof statin drugs have received either research grants
cholesterol, but by 1966 it was also apparent thator honoraria from Pfizer, Merck, AstraZeneca,
lowering cholesterol did not translate into a reducedNovartis, Glaxo Smith Kline, Johnson & Johnson,
risk of death from heart disease.Bayer, and many other drug companies that produce
As there was so much money to be made fromstatin drugs.
pharmaceutical development, the campaign toYou would think that with all the advertising and
produce cholesterol-lowering drugs kicked into highrecommendations from medical experts on the
gear, despite the lack of evidence showing that thebenefits of statin drugs, the medical community
lowering cholesterol reduced the risk of untimelywould possess overwhelming evidence that the drugs
death from heart disease.reduce the risk of death from cardiovascular disease.
Heart disease kills 725,000 Americans annually, withA hint of some of the smoke and mirrors in the
women accounting for 2/3 or nearly 500,000 ofpharmaceutical companies' advertising can be seen in
those deaths. After thirty years oftheir TV commercials. Read carefully the small print
cholesterol-lowering medications' failure to significantlyon some of Crestor's® commercial advertising.
lower the death rate from cardiovascular disease, inTheir commercial states how much it lowers LDL
1987 a new and more dangerous class of drugs wascholesterol. However, in the same ad you can read,
unleashed upon the world: the "statin" drugs."...Crestor® has not been shown to reduce the
Cholesterol-lowering statin drugs are now therisk of heart disease or heart attack." If so, then
standard of care that physicians are indoctrinated intowhy take it? Isn't the bottom line to prevent death?
prescribing to reduce cardiovascular disease. AreThe system for reporting adverse effects from
statin drugs the best way to prevent heart attacksmedications is tremendously flawed, so much so that
and death?many people are seriously harmed or killed by some
Before 1936 the most common type of heartmedications before they are finally removed from
disease was congestive heart disease (CHD). It rarelythe market. Most doctors do not know what
caused sudden death and could be treated with thesymptoms or effects are due to the drug, what
drug digitalis. The incidence of CHD remained stableshould be reported, or even to whom to report
until 1987, after which the incidence of the diseaseadverse effects. They assume that the research
skyrocketed. Interestingly, the timing of thethat went into developing the drug has already
increased incidence of congestive heart diseaseidentified all the effects and that a drug brought to
coincides with the introduction of cholesterol-loweringmarket is "safe." However, only one in twenty side
statin drugs. Could cholesterol-lowering statin drugseffects is ever reported to either hospital
have something to do with the weakening of heartadministrators or the FDA.
muscles and the increased incidence of congestiveStatin drugs block cholesterol production in the body
heart failure? We will see that lowering the body'sby inhibiting the enzyme called HMG-CoA reductase in
co-enzyme Q10 levels, a side effect of statin drugs,the early steps of its synthesis in the mevalonate
does indeed increase the risk of muscle damage,pathway. Cholesterol is one of three end products in
including the muscles of the heart.the mevalonate chain. This same biosynthetic
Atherosclerosis is a disease characterized primarily bypathway is also used to create co-enzyme Q10, or
inflammation of the arterial lining caused by oxidativeco-Q10, as well as dilochol. Therefore, one
damage from homocysteine, a toxic amino acidunfortunate consequence of statin drugs is the
intermediary found in everyone. Homocsyteine, inunintentional inhibition of both Co-Q10 and dilochol
combination with other free radicals and toxins,synthesis.
oxidizes arteries, LDL cholesterol, and triglycerides,The drug information insert of a statin drug states
which in turn releases C Reactive Protein (CRP) fromthat it lowers co-enzyme Q10 levels. Most doctors
the liver-a marker of an inflammatory response withinhave forgotten their biochemistry class in medical
the arteries. Inflammation (oxidation) is the beginningschool, and forgotten about the importance of
of plaque buildup and ultimately, cardiovascularCo-Q10. Therefore they apparently are not
disease. Plaque, combined with the thickening ofconcerned about such a statement on the drug
arterial smooth muscles, arterial spasms, and clotting,labeling information sheet. They may even reassure
puts a person at a high risk of suffering heart attacktheir patients that lowering Co-Q10 is nothing to
or stroke.worry about, but at the same time warn them that
For years, doctors have hyper-focused on cholesterolthe drug may cause liver damage and to have their
levels. First it was the total cholesterol; later theliver enzymes checked every three to six months to
focus became the ratio of "good" HDL cholesterol tomake sure the drug isn't killing them. They do not
"bad" LDL cholesterol. In other words, how much ofrealize that it is the depletion of Co-Q10 that leads to
your cholesterol was good, and how much was bad?liver damage and death.
Of the two, the important parameter is the level ofUbiquinone, or co-enzyme Q10, is a critical cellular
HDL cholesterol, not LDL cholesterol. HDL, ornutrient created in the cell's mitochondria, the
high-density lipoprotein cholesterol, is responsible for"engines" that produce energy for the cell.
clearing out the LDL cholesterol that sticks to arterialMitochondria use sugar, oxygen, and water to
walls. Exercise, vitamins, minerals, and otherproduce energy molecules known as ATP. Without
antioxidants, particularly the bioflavonoid and oliveATP cells could do nothing. Damaged tissues could
polyphenol antioxidants, increase HDL cholesterolnot be repaired. Cells could not divide or produce or
levels and protect the LDL cholesterol from oxidativeutilize proteins, enzymes, or hormones. Death of cells,
damage, and therefore do more to reduce the riskand indeed of the human body would occur if ATP
of heart disease than any medication ever could.could no longer be produced and utilized. Co-Q10
There is nothing inherently bad about LDL cholesterol.functions within the mitochondria as an electron
LDL cholesterol is critical to maintain life. LDLcarrier to cytochrome oxidase, our main respitory
cholesterol only becomes "bad" when it is damaged,enzyme, which helps turn oxygen and sugar into
or oxidized by free radicals. Only the damaged, orenergy. The heart requires high levels of oxygen,
oxidized form of LDL cholesterol sticks to the arterialsugar, and Co-Q10 since it utilizes a lot of energy. A
walls to initiate the formation of plaque.form of Co-Q10 called ubiquinone is found in all cell
Let us look towards cigarette smoking for a simplemembranes, where it plays a role in maintaining
example demonstrating that we really need tomembrane integrity, so critical to nerve conduction
reduce oxidized LDL cholesterol to preventand muscle contraction. Co-Q10 is also vital for the
atherosclerosis, as opposed to indiscriminatelyformation of elastin and collagen, which make up the
lowering LDL cholesterol with statin drugs. Everyoneconnective tissues of the skin, musculature, and the
knows that cigarette smoking increases the risk ofcardiovascular system.
many chronic diseases, such as cancer, heart disease,The most common side effect of statin drugs is
and stroke. Smokers with normal levels of LDLmuscle pain and weakness. In fact, many patients
cholesterol are at an even greater risk of developingwho start on the statin drugs almost immediately
heart disease than a non-smoker who has elevatednotice generalized fatigue and muscle weakness. This
levels of LDL cholesterol. Of course the reason whyis due to the depletion of Co-Q10 needed to support
a smoker with normal levels of LDL cholesterol is atmuscle function. Dr. Beatrice Golomb of San Diego,
greater risk of disease is because his LDL getsCalifornia, is currently conducting a series of studies
excessively oxidized.on statin side effects. The pharmaceutical industry
Cigarette smoke releases so many toxins and freeinsists that only 2-3% of patients get muscle aches
radicals that the LDL cholesterol, the triglycerides, andand cramps, when in fact in one study, Golomb found
the arterial walls are extensively oxidized.that 98% of patients taking Lipitor®, and
Homocysteine levels are also increased by cigaretteone-third of the patients taking Mevacor® (a
smoking which further oxidizes LDL cholesterol andlower dose statin), suffered noticeable to significant
the arterial lining. Oxidation is the initiating cause ofmuscle problems.
atherosclerosis. Therefore, the more and longer oneSome people on statin drugs lose coordination of
smokes, the more oxidative damage he sustains andtheir muscles. Some develop pain in their muscles,
the greater his risk of developing heart disease. Thesome are not able to write due to loss of fine motor
degree of oxidation directly corresponds to the riskskills. Many lose the strength to exercise. Others are
of heart disease.falling more frequently as their muscles give out, still
If you are not taking vitamins, minerals, andothers have trouble sleeping due to muscle cramping
antioxidants then your LDL cholesterol is beingand twitching. Even worse, many people are
oxidized, it is sticking to your arterial walls, and youexperiencing most of these side effects. The
ARE developing heart disease EVEN IF YOURproblems are so numerous, it is difficult to list all the
CHOLESTEROL LEVELS ARE NORMAL! LDLsymptoms people might experience. These problems
cholesterol starts sticking to arterial walls before thedo not come from the "disease" of high cholesterol,
age of 5.but the disease of ignorance in prescribing these
Among the many free radicals that damagedrugs.
cholesterol, triglycerides and the arterial lining isAs we age, Co-Q10 levels decline naturally. From the
homocysteine, a toxic intermediate biochemicalage of 20 to 80, Co-Q10 levels fall by nearly 50%.
produced during the conversion of the amino acidAlong with the natural decline of Co-Q10, comes a
methionine into another important amino acid,natural decrease in energy and an increase in the risk
cysteine. Both methionine and cysteine are non-toxic,of heart disease, stroke, and cancer. If the natural
but homocysteine is very toxic to the lining of thedecline of Co-Q10 levels increases the risk of fatigue,
arterial endothelium. Homocysteine oxidizes LDLcancer, heart disease, and stroke, would it not make
cholesterol, triglycerides and the arterial lining.sense that accelerating the decline of Co-Q10 levels
Homocysteine is an amino acid normally produced inwith statin drugs would have the same effect? They
small amounts from the amino acid methionine. Thedo indeed!
normal role of homocysteine in the body is to controlDemonstrating the importance of Co-Q10 to
growth and support bone and tissue formation.cardiovascular health, in a randomized, double blind,
However a problem arises when homocysteine levelsplacebo-controlled study of people either taking or
in the body are elevated, causing excessive damagenot taking statin drugs, supplementation with Co-Q10
to LDL cholesterol, as well as to arteries.reduced the risk of heart attacks and death in those
Furthermore, homocysteine actually stimulateswith heart disease and prior heart attacks by 50%,
growth of arteriosclerotic plaque, which leads toregardless of whether they were on a statin drug or
heart disease.not. (Singh R, Neki N, Kartikey K, et al. Effect of
Thyroid hormone controls the level of homocysteine,coenzyme Q10 on risk of atherosclerosis in patients
but numerous factors play a role in the elevation ofwith recent myocardial infarction. Mol Cell Biochem.
homocysteine. Normal aging, kidney failure, smoking,2003 Apr; 246(1-2):75-82.)
some medications, and industrial toxins all elevateAdditionally, Co-Q10 was shown to increase blood
homocysteine levels. Interestingly, estrogen helpslevels of vitamin E and significantly increase the levels
lower homocysteine.of protective HDL. As low HDL is a major risk factor
Homocysteine becomes elevated in the blood with afor heart disease, increasing it is a definite benefit.
deficiency of the B vitamins-B6, B12 and folic acid.Statin drugs were shown not to provide any benefit
Genetics also play a role. About 12% of thebeyond that of supplementing with Co-Q10. Let me
population has an undetected defect requiring highermake this clear - in this study only the co-enzyme
levels of folic acid than the rest of population to helpQ10 provided any benefit, not the drugs!
maintain homocysteine levels in a safe range (belowCardiologist Dr. Peter Langsjoen of East Texas
6.5). Therefore if you have high homocysteine levelsUniversity reported the effects of Lipitor®
(> 7.0) even though you are taking supplemental Bamong 20 patients who started with completely
complex vitamins, then you may be among the 12%normal hearts. After six months on a low dose of 20
who need more than 1000 mcg of folic acid per day.mg of Lipitor® per day, two thirds of the
In addition, betaine, also known as trimethylglycinepatients started to show signs of heart failure, as
(TMG) lowers homocysteine.seen by abnormalities in the heart's filling phase.
Homocysteine is second only to cigarette smoking inAccording to Dr. Langsjoen, this malfunction is due to
its oxidative destruction. It causes small nicks or tearsCo-Q10 depletion. Nine controlled trials using statin
in the arterial lining, while also oxidizing and damagingdrugs in humans have been conducted thus far. Eight
LDL cholesterol. The damaged, or oxidized LDLof these showed significant statin-induced Co-Q10
cholesterol sticks to the homocysteine-damageddepletion leading to a decline in left ventricular
areas of the arterial lining. The combination offunction and other biochemical imbalances.
oxidized LDL cholesterol and a damaged arterial liningIn the United States, the incidence of heart attacks
is what causes LDL cholesterol to stick to theover the past ten to fifteen years has declined
arteries, whether or not the LDL cholesterol level isslightly. But congestive heart failure and
normal.cardiomyopathy have risen alarmingly. Is it a
Cholesterol-lowering statin drugs are the standard forcoincidence that statin drugs were first marketed in
treating high cholesterol. This is dogma, and anyone1987, and then from 1989 to 1997, deaths from
who states otherwise is committing medical heresy.congestive heart failure more than doubled? 38 It
Many people find it hard to believe thatscares me that virtually all patients with heart failure
pharmaceutical companies could ever succeed inare put on statin drugs, even if their cholesterol is
paying medical researchers, medical associations, andalready low. In my opinion, the worst thing to do for
doctors to recommend something detrimental to oura failing heart is take a statin drug. The best thing is
health.to take is a full range of quality nutritional
Most people do not know that pharmaceuticalsupplements, ...vitamins, minerals, fish oil, and other
companies fund medical institutions, medical education,antioxidants, including Co-Q10.
medical conferences, and still reward doctors andVarious antioxidants work synergistically, each
research institutions for providing favorable results oncontributing to the fight against free radicals in
their drugs. Likewise, pharmaceutical companies oftendifferent areas and in different ways. In the blood
suppress negative results from studies done on theirstream, water-soluble antioxidants, such as vitamin C,
drugs. Money has the power to sweep negativeand grape seed extract come in contact with and
results and serious side effects under the rug. Moneyneutralize free radicals before they damage
has the power to influence the FDA to decide whichLDL-cholesterol. Other antioxidants saturate arterial
drugs make it to market and which drugs becomewalls and other tissues, and protect collagen and
the "standard" of treatment.elastic fibers from free radical damage, reducing
Former editor of the New England Journal of Medicineinflammation and plaque formation. The fat-soluble
(NEJM), Dr. Marcia Angell, warned of the problem ofantioxidants, vitamin E, beta carotene, and
commercializing scientific research in her outgoingco-enzyme Q10 ride along in the blood fat
editorial titled "Is Academic Medicine for Sale?" Angell(triglycerides) and LDL cholesterol, protecting them
called for stronger restrictions on pharmaceuticaland the endothelium from oxidation. Vitamin E sits on
stock ownership and other financial incentives forthe surface of LDL cholesterol, protecting it from
researchers. She said that growing conflicts offree radical damage. Beta carotene, grape seed
interest were tainting science, warning "When theextract and olive extract penetrate deeper inside the
boundaries between industry and academic medicineLDL cholesterol and arterial walls, adding more
become as blurred as they are now, the businessprotection from oxidation. Quercetin and alpha lipoic
goals of industry influence the mission of medicalacid work through nitrous oxide pathways to reduce
schools in multiple ways." She did not discount thehigh blood pressure, a major risk factor for heart
benefits of research but said, "a Faustian bargain"disease.
now existed between medical schools and theA report published in the Archives of Internal
pharmaceutical industry. Angell left the NEJM in JuneMedicine in 2005 looked at 97 double-blind controlled
2000 and has written a book, "The Truth About thestudies comparing the efficacy of cholesterol-lowering
Drug Companies: How They Deceive Us and What tostatin drugs to fish oil. They found that
Do About It."cholesterol-lowering statin drugs reduced the risk of
Two years later, in June 2002, the NEJM announceddeath from heart disease by only 13%, andinteresting
that it was going to begin accepting articles thatenough it was NOT due to the effect of lowering
were written by biased researchers, as there weren'tcholesterol. The benefits, although small, were
enough unbiased researchers left to write articles. Inderived from the fact that statin drugs have a slight
other words, most research institutions were nowantioxidant effect.
funded by one or more of the numerousEven more interesting, the salmon oil was shown to
pharmaceutical companies.reduce the risk of death from heart disease by 23%,
An ABC report noted that a survey of clinical trialsnearly double the benefit of statin drugs. Salmon oil is
revealed that when a drug company did not fund aan omega-3 fatty acid that gets incorporated into
study, favorable results regarding a drug were foundcholesterol and triglycerides and prevents the
only 50% of the time. In studies funded by drugoxidation of LDL cholesterol. Since LDL cholesterol is
companies favorable results about the drugs wereprotected from excessive oxidation there is less
reported an amazing 90% of the time. Money canplaque buildup and less risk of heart disease.
and does buy the desired results. This is how mostInflammation is a well-known component in the
medical research and drugs are now developed andformation of atherosclerosis. To keep it simple, think
brought to market.of inflammation and oxidation as the same process.
In 1977, the internationally-renowned heart surgeon,The immune system's response to inflammation is
Dr. Michael DeBakey pointed out that only 30-40% oftorelease peroxides that act like acid to break down
people with blocked arteries and heart disease havedamaged tissues, so that cells from the immune
elevated blood cholesterol levels, and posed thesystem, macrophages, can consume the molecules
logical question, "How do you explain the otherand clean up the site. But peroxides escalate the
60-70%?"oxidation/inflammation process, thus damaging more
Because lowering cholesterol did not reduce the risktissue. The arterial walls become more inflamed,
of death from heart disease, the Cholesterolescalating the formation of plaque and scarring. The
Consensus Conference in 1984 developed newdownward cycle continues until atherosclerosis is so
guidelines to lower the "acceptable level" ofadvanced that the occurrence of a heart attack or
cholesterol. High cholesterol would now be thestroke becomes imminent.
diagnosis for any man or woman with a cholesterolThe liver's response to inflammation is to release C
level over 200. Doctors had to convince their patientsreactive protein (CRP) into the blood. Other
that they had the disease and needed to take oneinflammatory causes can cause elevated CRP levels,
or more expensive drugs for the rest of their lives.including cigarette smoking, obesity, insulin
However, when lowering total cholesterol levelsinsensitivity, diabetes, rheumatoid arthritis, infections,
below 200 did not translate into saving lives fromdementia, colorectal cancer, high blood pressure, and
heart attacks, the focus then turned to LDLaging. Accordingly, elevated CRP levels are a direct
cholesterol levels. The "disease" of high cholesterolindication of inflammation in the body and that
was refined to the disease of high LDL cholesterol.atherosclerosis, including heart disease, is actively
The unfortunate patient who had an LDL cholesteroldeveloping.
level above 130 was now condemned to a lifetime ofHomocysteine and high sensitivity CRP levels can and
expensive drugs. Though completely illogical, evenshould be tested. Dr. Jialal, of the Universtity of
when a person with normal LDL cholesterol levelsTexas Southwestern Medical School at Dallas, is well
suffered a heart attack, he would still be prescribed aknown for his research correlating oxidized LDL
cholesterol-lowering drug.cholesterol as the true cause of atherosclerosis, has
As we shall see, statin drugs reduce the risk of deathalso identified high sensitivity C reactive protein as a
by repeat heart attacks by as much as 30%, butpredictive risk factor for inflammation of arterial walls
interestingly enough, the mechanism of action inand plaque formation. Your doctor may not test for
reducing the risk of death after a heart attack is notthese routinely, but you should insist on getting these
via statin drugs' ability to lower cholesterol! It hastests done. Both of these predictive values can be
been discovered that statin drugs have a modestkept at "safe" levels. Vitamins, minerals, antioxidants,
anti-inflammatory and antioxidant effect. Yet, thereand omega-3 fatty acids can lower the levels of
are many natural antioxidants that reducehomocysteine and CRP. The B vitamins, along with
inflammation and oxidation of LDL cholesterol and thebetaine, or tri-methyl-glycine (TMG), change
lining of the arteries, which may soon be discoveredhomocysteine into safer amino acids and reduce
to be more effective in reducing the risk of deathinflammation of the LDL cholesterol and the arterial
than "antioxidant drugs," without toxic side effects.lining.
The myth that high LDL cholesterol is the primaryWhen you receive the results of your homocysteine
cause of heart disease, and that we must be ontest, do not accept the answer, "Your test was
drugs to protect ourselves is dispelled by thenormal." Ask for the actual number. The doctor and
evidence. If the premise were true that people withnurse usually know what is normal by what the lab
high levels of LDL cholesterol get heart disease, thenslip states as the "normal range." Most lab results
we could assume that people with normal levels ofreport a normal homocysteine level as being below
LDL should not get heart disease, or at least very10.4, when in fact, since the early 1990's, researchers
few should get it. However, as Dr. DeBakeyhave known that a homocysteine count above 6.5
observed, approximately 60% of those who diesignals a rapid linear rise in the risk for heart disease.
from heart disease have normal LDL cholesterolFurthermore, with every 3 point elevation of
levels!homocysteine above 6.5, e.g., when homocysteine
Furthermore, after over 45 years of doctorslevels are 9.5, the risk of coronary artery disease
prescribing cholesterol-lowering drugs, heart disease(CAD) rises by an additional 35%! Yet you may be
and stroke still remain the number one cause oftold that 9.5 is "normal and not to worry." With a
death in both women and men. This says thathomocysteine level of 12.5, the increase in therisk for
regardless of whether you have a high or a normalheart disease exceeds 70%. The greater the
level of cholesterol, you have a 50% chance of dyinghomocysteine level, the greater the oxidationof both
from heart disease. If this is so, and it is, then whyLDL cholesterol and the arterial lining. The greater the
take a dangerous drug to attempt to lower yourinflammation, the higher the CRP. Is it any wonder
cholesterol in the first place?that homocysteine and CRP levels are more
In 2001, the target level of LDL cholesterol waspredictive for risk of heart disease than cholesterol
lowered from 130 to 100, and overnight the numberlevels and ratios?
of people considered to be candidates for cholesterolI need to emphasize that anyone whether they have
statin drugs doubled. Many people such as myselfa medical problem or not, should discuss this
bristled at the news, because we knew theinformation with their physician before acting upon
effectiveness of vitamins, minerals, and antioxidantsanything written here. The information provided is not
in preventing and reversing heart disease. Many of usmeant to diagnose or treat any disease. It is for
could see the conspiracy for what it was.informational purposes only; and no one should make
The level at which LDL cholesterol is considereddecisions about their medications without consulting
normal has continually been influenced bywith their physician. No one should come off a
pharmaceutical companies, who pull the financialcholesterol-lowering statin drug in lieu of nutritional
strings of research grants that keep medical schoolssupplements without a thorough discussion with their
and medical organizations in business. The lower theyphysician who is keenly aware of all the pros and
can establish the level at which LDL cholesterol iscons of both treatment modalities.